CLL /Chronic Lymphocytic Leukemia/
Assorted Experiences with CLL
Male patient (42); diagnosed on 02-02-2006; on Dd-water from 02-18-2006
Chronic lymphocytic leukemia (CLL) was diagnosed in February 2006. At that time the white blood cell (WBC) count was 13.6 G/L, and enlarged lymph nodes were verified in the axilla, neck and inguinal region. Dd-water consumption was initiated upon diagnosis. One month later the WBC count started decreasing and by the 7th month of the cure the WBC count reached the physiological level. In addition, the platelet count increased from 100 G/L to 150 G/L. As a result of the Dd-water consumption, the enlarged lymph nodes became softer and continuous decrease in their size was also observed. Enlargement of the spleen was verified at the diagnosis, but one year later the size of the spleen was determined to be normal. To sum up, during the last one and a half year of Dd-water consumption there was a significant improvement in the patient’s condition and not any forms of conventional therapy was applied.
Male patient (61); diagnosed on 09-15-1996, on Dd-water from 11-15-2001
The patient was diagnosed with CLL in 1996 (WBC count: 24 G/L). Conventional oncological treatments were not applied during the first three years of the illness. Chlorambucil treatment was initiated in autumn 1999 because the WBC count was 60 G/L. The chlorambucil treatment was still running on a weekly base, when the patient started consuming Dd-water in November 2001. In four months, the enlarged lymph nodes became smaller and ten months later there were no palpable lymph nodes. In November 2002 the WBC count fell to 20 G/L. The patient extended the Dd-water consumption until June 2003, but further decrease in WBC count was not detected. The examination in 2005 verified stagnation of the WBC count and complete regression of the status of lymph nodes. In summary, consumption of the Dd-water contributed to the effects of conservative forms of treatment. Eleven years after the diagnosis, the patient is free of complaints and symptoms.
Male patient (64); diagnosed on 06-15-2006; on Dd-water from 03-15-2007.
The patient was diagnosed with CLL in June 2006. In March 2007, when he started consuming Dd-water, the enlarged lymph nodes in the inguinal region on either side caused pain in motion. Lymph nodes in the armpits, neck, head and face were conspicuously swollen. WBC count was 30 G/L and increased continuously. During the first three months the patient consumed Dd-water with a D-content of 105 ppm, but the WBC count showed not significant changes (05.24. 2007: 30.9 G/L, 06. 12. 2007: 30.1 G/L). The axillary lymph nodes became softer. The patient was advised to change to Dd-water containing D at a lower concentration of 85 ppm. The next blood screening showed remarkable decrease of the WBC count (07-15- 2007: 26 G/L) and the process is still continuing (09-13-2007: 11,000 G/L). His pain in the leg subsided. In summary, D-depletion in its own stopped the continuous increase in WBC count and then resulted in a significant decrease. Although the enlarged lymph nodes are still present, the patient does not complain of any symptoms.
Male patient (64); diagnosed on 10-15-1995; on Dd-water from 01-18-1996.
Because of a swollen lymph node on the neck the patient underwent aspiration cytology in 1992, but malignant process was not verified. CLL was diagnosed in 1995 with a WBC count of 131 G/L, and numerous enlarged lymph nodes were verified with chest CT scan and abdominal ultrasonography. The enlargement of the spleen and liver was also remarkable. Although the patient was given chemotherapy with chlorambucil, bone marrow infiltration was 95 % in January 1996. The patient received blood transfusions weekly, he was in a weakened condition and had lost weight, when he started consuming Dd-water. During the next two months blood transfusion was given only twice. In additional two months the lymph node on the neck was no longer palpable and the patient was doing gardening. Abdominal symptoms developed in November 1996 caused by the palpable conglomerate of enlarged lymph nodes. As a result of the high-dose chlorambucil and prednisolone the symptoms transiently subsided, but in January 1997 the patient had recurrence of symptoms and subsequently underwent abdominal surgery, when the necrotized lymph nodes were excised. The patient did not restored the Dd-water consumption. During the next year no progression occurred, he enjoyed a good quality of life and gained weight. In March 1998 progression developed, and the patient died in October 1999. We can conclude, that the conventional form of treatment was not effective, but D-depletion resulted in significant improvement of the patient’s status. The patient died almost four years after the bone marrow infiltration was 95 % high despite of the intense conventional treatment.
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